English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

This 53 year old white female presented with complaints of severe pelvic congestion syn-drome and hematuria. The patient gave a history of intermittent macro- and microscopic hematuria for the past 3 years. A recent urologic examina-tion with blue light cystoscopy had revealed some submucosal varices in the bladder, which were thought to be the source of bleeding. However, 2 cystoscopies, 1 and 2 years prior, had not shown any varices. The patient had a history of ethanol abuse and at 2 occasions experienced hematemesis which was controlled by application of a Black-more tube. A gynecologic examination comple-mented by ultrasound revealed a 3 cm submucosal fibroid. Laboratory finding were HB 12.8, HCT 38, RBC 4.2 mil, Platelets 180000, WBC 5800, BUN 28, Creatinine 1.3, estimate GFR 42, Na 136, K 5.6; Cl 98 mEq/lt, alk pts 18 KA U, Tot. Bil 0.6; Glucose 107, PT 10.0 , INR 1.3, Transaminase 500U. Urine-analysis revealed a spec grav 1022, 12 RBC/ hpf, 1.2 WBC/hpf, isolated clumps of bac-teria; culture showed no growth. Physical exami-nation noted pale complexion, distended abdomen with palotment suggesting the presence of ascitis; the spleen was markedly enlarged, BP 138/82, PR 78, Respiration 20 and regular. An enhanced angioCT demonstrated massive esophageal and gastric fundus varices (Figure-1). The spleen was markedly enlarged. There were spleeno-renal shunts resulting in mas-sive dilatation of the left renal vein (Figures 2,3). The left renal vein dilated to a degree, to be com-promised in its passage between aorta and supe-rior mesenteric artery (Figure-3). Dilated collateral veins to the umbilicus were present. In the pelvis one notes massive dilatation of parametrial veins; pelvic congestion syndrome (Figure 4).Renal failure in cirrhotic patients is usu-ally attributed to “hepato-renal syndrome” a con-sequence of albumen loss to ascitis. In our patients the massive spleno-renal shunt caused increased flow in the left renal vein and thereby increased left renal vein pressure (1). This was probably the cause for microscopic hemarturia that had been observed for 3 years (Figure-3). Moreover, the in-creased renal vein pressure caused parenchymal

Microscopic hematuria and pelvic congestion syndrome in a patient with cirrhosis