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Chagas’ disease has marked epidemiological importance, even with the significant reduction of its transmission, due to the contingent of infected individuals with potential for severe forms of development1. Chronic chagasic cardiopathy is the most serious manifestation of the disease, constituting an important cause of heart failure in Brasil2. Approximately 30 to 40% of infected individuals will present some degree of cardiac involvement during their life3. Chagasic cardiomyopathy (MCh) presents a variable clinical course, with a worse prognosis in comparison to other myocardiopathies. The involvement of the right ventricle represents a peculiar characteristic of Chagas’ disease, especially when described in the early stages of the disease4. However, few studies have evaluated the functional and anatomical conditions of the right ventricle in MCh, compared to idiopathic dilated cardiomyopathy (MDI). Right ventricular function is determined by the intrinsic contractility of this chamber and ventricular preload and afterload conditions. In MCh, VD dysfunction can be attributed to the inflammatory process itself, secondary to the infection by the parasite, causing depression of its contractility, but without clinical manifestations. Some abnormalities are not detectable through conventional echocardiographic techniques, requiring more accurate techniques to analyze their contractility. With the elevation of pressure in the pulmonary artery, secondary to left ventricular dysfunction, there is an increase in the post-load, favoring dysfunction with clinical evidence of systemic congestion5. The prognostic value of impaired right ventricular function was also verified in ischemic and non-ischemic heart failure6. Right ventricular dysfunction was associated with mortality6. Lewis et al. 1993, after studying 67 patients with MDI, found that the right ventricle area was the only significant predictor of death. In addition, a study of a subgroup of 205 more stable patients with moderate heart failure showed that the prognostic value was independent of right ventricular function7. Regardless of the cause of myocardiopathy, LV function contributes to VD performance through septal contractility. The interventricular septum represents an integral component of the VD architecture and mechanics. Under physiological conditions, septal contraction constitutes 1/3 of the VD contraction, evidencing the systolic interaction between the ventricles8. Ghio et al. 2001, studying 377 patients with heart failure and significant left ventricular dys-

Right ventricular function in dilated cardiomyopathies with chagasic and idiopathic etiologies