English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Received: May 15, 2019; Accepted: June 05, 2019; Published: June 10, 2019 In our “macroscopic” world it is a truism that an action is often followed by an equal and opposite reaction. In the “microscopic” realms (chemical, biological, quantum environments), that response often is mimicked. For example, stressed cancer cells can exhibit enhanced resistance to therapy and development of alternate genetic/epigenetic responses that increase proliferation and developmental / proliferative events unanticipated by the physician. These considerations provide a background for considering these and other possible consequences to hypoxia or other cellular stresses by deterministic, or by random, stochastic cancer cell responses to stresses. According to some, random refers to a variable, stochastic to a process or system.

If the statistics underlying macroscopic and microscopic (quantum scale) events were to fundamentally differ would this affect the biology of cancer?