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a pregnant horn of the uterus increased in diameter to a mean value of 167% of its diameter in non-pregnancy. In cases ofunilateral pregnancy, the uterine artery supplying the non-pregnant horn of the uterus was not increased in diameter. Administration of atropine (0\m=.\1to 0\m=.\5mg/kg) to animals in late pregnancy caused constriction of the enlarged uterine arteries to a mean value of 137% of their pre-pregnant diameter, but the diameters of the internal iliac and inferior epigastric arteries were not changed. In non-pregnant animals, the diameter of the uterine arteries was not affected by atropine. The results support previous evidence for an effector r\l=o^\leof cholinergic vasodilator nerves in the production of uterine hyperaemia during pregnancy in the guinea-pig.

ARTERIOGRAPHIC EVIDENCE FOR A CHOLINERGIC DILATOR MECHANISM IN UTERINE HYPERAEMIA OF PREGNANCY IN THE GUINEA-PIG