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References: 1. Tierney, T. et al. Evidence from studies on co-cultures of TtT/GF and AtT20 cells that annexin 1 acts paracrine or juxtacrine mediator of the early inhibitory effects of glucocorticoids on ACTH release. Journal of Neuroendocrinology, 15, 1134-1143 (2003) 2. Morris, J.F. et al. Lack of annexin 1 results in an increase in corticotroph number in male but not female mice. Journal of neuroendocrinology, 18, 835-846 (2006) 3. Ye, R.D. et al. International union of basic and clinical pharmacology. LXXIII. Nomenclature for the formyl peptide receptor (FPR) family. Pharmacological reviews, 61, 119-161 (2009) 4. Hartt, J.K et al. N-formylpeptides induce two distinct concentration optima for mouse neutrophil chemotaxis by differential interaction with two N-formylpeptide receptor (FPR) subtypes: Molecular characterization of FPR2, a second mouse neutrophil FPR. Journal of Experimental Medicine, 190, 741-747 (1999) 5. Dufton, N. et al. Anti-inflammatory role of the murine formyl-peptide receptor 2: Ligand-specific effects on leukocyte responses and experimental inflammation. Journal of Immunology, 184, 2611-2619 (2010) 6. McArthur, S. et al. Annexin A1 regulates hormone exocytosis through a mechanism involving actin reorganization. FASEB Journal, 23, 4000-4010 (2009) 7. Solito, E. et al. A novel calcium-dependent proapoptotic effect of annexin 1 on human neutrophils. The FASEB journal, 17, 1544-1546 (2003) 8. McNicol, A.M. et al. The mitogenic effects of corticotrophin-releasing factor on the anterior pituitary gland of the rat. Journal of Endocrinology, 118, 237-241 (1988) Introduction

Lack of Fpr2/Fpr3 receptors alters the structure and function of pituitary corticotrophs