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Sex steroids are important for the maintence of the female and male skeletons. Androgen deficiency is one of the major factor leading to the development of osteoporosis in men. Another important hormone involved in bone metabolism is calcitonin (CT). This potent hypocalcemic peptide, produced by thyroid C cells, contributes to calcium (Ca) homeostasis by direct inhibition of osteoclast-mediated bone resorption and output of Ca from skeletal tissues. Since CT is a potent antiresorptive agent In this study we investigated the effects of tamoxifen (TAM), as a selective estrogen receptor modulator on CT-producing thyroid C cells, skeletal and hormonal changes in middle-aged orchidectomized (Orx) rats as an animal model of male osteoporosis. Effects of the tamoxifen on calcitonin producing th yroid C cells and bone in rat model of male osteoporosis

Effects of the tamoxifen on calcitonin producing thyroid C cells and bone in rat model of male osteoporosis