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Alzheimer's disease (AD) is characterized by extracellular deposition of β-amyloid (Aβ) as senile plaques, as well as intracellular accumulation of hyperphosphorylated, aggregated tau as neurofibrillary tangles. Increased production and/or deposition of Aβ is thought to precede tangle formation,

Linking Amyloid and Tau Pathology in Alzheimer's Disease: The Role of Membrane Cholesterol in Aβ-Mediated Tau Toxicity: Figure 1.