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Interneurons mediate inhibition in the CNS by releasing GABA, which hyperpolarizes postsynaptic neurons by promoting chloride entry through GABAA receptors. Low levels of intracellular chloride ([Cl−]i) in postsynaptic neurons, maintained by the chloride exporter KCC2, are required for the

A Potential Role for Nerve Growth Factor in Regulating the Maturation of Inhibitory Neurotransmission