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Its mechanism of action is manifold, with recent published studies recognizing: inhibition of osteoclastic bone resorption, induction of osteoblasts to the secretion of an osteoclastmediated inhibition of bone formation, induction of apoptosis in osteoclast (“programmed cell death”), an antiangiogenic effect that reduces endothelial growth factor (EFG). Also, associated with the previous ones, there is an alteration of the macrophages and the cicatrization of the oral mucosa by involvement of the fibroblasts and keratinocytes [2].

journal of rheumatology and arthritic diseases

A Rare Case of Epidermoid Carcinoma on Mandibular Osteonecrosis Due to Oral Bisphosphonates in the Treatment of Osteoporosis. Case Report and Review of Literature