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fiber pennation angle) but also by its neurophysiological properties (e.g., motor unit recruitment, rate coding, motor unit synchronization). Several studies have shown that the deficits in knee extensor strength found after knee injuries are attributable more to decreased knee extensor muscle activation than to muscle atrophy due to disuse [1-3]. The inability to maximally activate the knee extensors in the absence of frank tissue damage can alter athletic performance, which often relies on optimal contraction of this lower-extremity muscle group [1].

Clinical Measures Predicting Knee Extensor Muscle Activation During a Maximal Voluntary Isometric Contraction