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The effective treatment of DM and the prevention of diabetic complications may be improved by a better understanding of the antioxidant function of intracellular defences against (OS) stress. Polymorphisms in antioxidant genes may determine cellular OS levels as a primary pathogenic role in DM and/or in its complications. SOD-2 has been investigated in patients with several diseases, including type 1 DM (T1DM) to ascertain if specific genotypes have any protective influences in the pathogenic mechanisms in DM and/or in several different complications, including retinopathy, nephropathy and diabetic controls compared to normal healthy controls. A focus on the SOD-2 mitochondrial targeting sequence (Ala -9 Val substitution) found to be important in diabetic nephropathy.

A Review of the Role of Mitochondrial Manganese Superoxide Dismutase in Human Disorders, such as, Diabetes