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O 3  is the major component of photochemical oxidants and gives rise to visible injuries on plant leaves. In O 3 -exposed plants, O -  2  is produced before the formation of the injury, but the role that O -  2  plays in plant response to O 3  exposure is still unknown. To clarify its role, we observed the behavior of plants during O 3  exposure after pretreatment with tiron, which is an O -  2  scavenger. When tiron-pretreated tobacco cv. Bel W3 was exposed to O 3 , leaf damage was attenuated. In O 3 -exposed tobacco, tiron inhibited increases in the levels of ethylene and salicylic acid, which promote leaf injury. Tiron pretreatment also suppressed increases in the expression of O 3 -induced genes. These results suggest that O -  2  is involved in many plant responses induced by O 3  exposure. Bel B, a tobacco cultivar that is genetically related to Bel W3, is reported to be more resistant to O 3  than Bel W3, but the reason for this difference is unclear. We investigated the differences between the responses of Bel B and tiron-pretreated Bel W3 to O 3  exposure, and we discuss the reasons for the resistance to O 3  by comparing the phenotype of Bel B with that of tiron-pretreated Bel W3.

O - 2 Activates Leaf Injury, Ethylene and Salicylic Acid Synthesis, and the Expression of O 3 -Induced Genes in O 3 -Exposed Tobacco