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Beta-2-mimetics are currently the drugs of choice for the treatment of premature labor. The presence of beta-2-adrenoceptors in numerous organs of the body and the limited receptor specificity of the beta-2-stimulant give rise to a number of cardiopulmonary and metabolic side effects. While most of these side effects are explicable there is no satisfactory explanation for the occasional development of maternal pulmonary edema. In the last years systematic experimental and clinical investigations of GROSPIETSCH and co-workers [2] and our group [3, 4] have elucidated the pathophysiology of cardiopulmonary changes during tocolytic treatment by betamimetics.

journal of perinatal medicine

Aspects of the pathophysiology of maternal lung edema during tocolytic therapy (Comment on: High permeability pulmonary edema (ARDS) during tocolytic therapy, by RUSSI et al.)