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For optimal monitoring of the fetus during birth, cardiotokography is used in modern obstetrics. Clinicians depend upon the recording of . cardiac frequency and uterine contractions for therapeutic decisions, in particular when this diagnostic methpd is complemented by microblood examinations. However, our knowledge about the pathophysiologic interrelations between fetal cardiac frequency patterns, hemodynamics and acid-basemetabolismis still quiteincomplete. While clinicians are familiar with the clinical significance of a "silent fluctuation" of the cardiotokography, its cause is unknown, the all-encompassing term "fetal asphyxia" notwithstanding. Therefore, we have attempted to simulate in our laboratory conditions of fetal distress in acute experiments on the term lamb fetus in order to investigate possible causal interrelations. Conditions of fetal distress can be caused by a decrease in uterine blood perfusion, e. g. during a contraction or with maternal shock or hypoxia. These conditions must simulate nature as closely as possible for the investigation of the consequences for the fetus of acute maternal-fetal metabolic disturbances. As other investigators, have we used the model of maternal hypoxia in our experiments with some modifications.

The effects of experimental acute decrease of uterine perfusion and maternal hypoxia on the fetus