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Objective:  Progesterone (P 4 ) has been clinically shown to prevent the recurrence of preterm birth. The mechanism(s) of action is unclear, but may involve modulation of the immunologic inflammatory response of the lower genital tract. We evaluated the effects of P 4  on interleukin-8 (IL-8) production by vaginal and cervical epithelial cells stimulated with bacterial species that are commonly associated with preterm birth.

Does progesterone inhibit bacteria-stimulated interleukin-8 production by lower genital tract epithelial cells?