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Alterations of the intrauterine and early postnatal nutritional, metabolic, and hormonal environment may cause predispositions to the development of disorders and diseases in later life. Mechanisms responsible for this perinatally acquired ‘malprogramming’ still remain unclear. It has long been known, however, that hormones are environment-dependent organizers of the developing ‘neuroendocrine-immune network’, which regulates all fundamental processes of life. When present in nonphysiological concentrations during critical ontogenetic periods, hormones can therefore also act as ‘endogenous functional teratogens’.

‘Fetal programming’ and ‘functional teratogenesis’: on epigenetic mechanisms and prevention of perinatally acquired lasting health risks