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Aims:  A-Kinase anchoring proteins (AKAPs) coordinate the specificity of protein kinase A signaling by localizing the kinase to subcellular sites. The 1936G (V646)  AKAP10  allele has been associated with adults with low cholinergic/vagus nerve sensitivity and with newborns with increased blood pressure. Decreased activity of the parasympathetic system is associated with risk of metabolic syndrome. The aim of this study was to answer the question of whether 1936A&gt;G  AKAP10  polymorphism is associated with metabolic changes in full-term newborns that are predictive factors for the metabolic phenotype in adulthood.

journal of perinatal medicine

Polymorphism 1936A&gt;G in the  AKAP10  gene (encoding A-kinase-anchoring protein 10) is associated with higher cholesterol cord blood concentration in Polish full-term newborns