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Background : In response to many stress stimuli, cardiomyocytes produce a common set of heat shock proteins (HSP). Up-regulation of HSP70-1 (the inducible isoform) is known to reduce the risk of myocardial cell damage during open-heart surgery and seems to be protective against ischemia. We assessed  hsp70-1  gene expression during blood cardioplegic arrest in children undergoing surgical correction of congenital heart defects.

Heat shock protein 70-1 gene expression in pediatric heart surgery using blood cardioplegia