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Background:  Gilbert’s syndrome is a common metabolic dysfunction characterized by elevated levels of unconjugated bilirubin in the bloodstream. This condition is usually caused by additional (TA) insertions in a promoter region of the uridine diphosphate glucuronosyltransferase 1A1 ( UGT1A1 ) gene, which instead of the sequence А(TА) 6 TАА contains А(TА) 7 TАА. While the condition itself is benign, it presents elevated risk for patients treated with irinotecan, a common chemotherapy drug.

Microarray with LNA-probes for genotyping of polymorphic variants of Gilbert’s syndrome gene UGT1A1 (TA)n