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We sought to study the effects of chronic exposure to fluoxetine - a selective serotonin reuptake inhibitor (SSRI) and specific 5-HT(2B) receptor agonist in astrocytes - on the expression of kainate receptors (GluK1-5) in cultured astrocytes and in intact brains in mice and on GluK2 editing by adenosine deaminase acting on RNA (ADAR), as well as the ensuing effects of fluoxetine on glutamate-mediated Ca(2+) influx and extracellular signal-regulated kinase (ERK)(1/2) phosphorylation in astrocytes.

journal of psychiatry and neuroscience

Fluoxetine affects GluK2 editing, glutamate-evoked Ca<sup>2+</sup> influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes

Fluoxetine affects GluK2 editing, glutamate-evoked Ca2+ influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes

Fluoxetine affects GluK2 editing, glutamate-evoked Ca²⁺ influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes