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It is known today that low grade inammatory mechanisms play a pivotal role in the pathogenesis of cardiometabolic diseases in- cluding the metabolic syndrome (1-4). In the same vein, in ad- dition to its role in cholinergic neurotransmission, acetylcholine exerts a potential anti-inammatory eect mediated by "cho- linergic anti-inammatory reex" (5). Accordingly, a positive correlation of the blood serum level of acetylcholinesterase, bu- tyrylcholinesterase and C-reactive protein (CRP) was reported in obese and diabetic patients (6-10). In the present study we measured serum levels of acetylcho- linesterase, an enzyme hydrolizing acetylcholine, and CRP, a known inammatory marker, in patients with mild stage (n=14) and with advanced stage (n=14) of metabolic syndrome, accord- ing to the criteria of the United States National Cholesterol Edu- cation Program's Adult Treatment Рanel III (ATP III). e con- trol group comprised of age- and sex-matched healthy subjects (n=7).

Serum levels of acetylcholinesterase in metabolic syndrome