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Nonconvulsive absence epilepsy and convulsive epilepsy seizures are rarely seen in the same patient. It has been demonstrated that there is a resistance to development of convulsive seizures in genetic absence epilepsy models. The present study investigated glutamic acid decarboxylase (GAD) immunoreactivity in the brain region related to the interaction of these two seizure types, namely substantia nigra pars reticulata (SNR) subregions, SNR anterior and SNR posterior .

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Evaluation of GAD67 immunoreactivity in the substantia nigra pars reticulata region of the genetic absence epilepsy rats for the resistance to development of convulsive epilepsy seizure