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Background Salivary glands’ diseases have a varied etiology that includes autoimmune, infectious, idiopathic, and hereditary causes. The unifying factor in these diseases is the formation of granulomas, which histologically are mononuclear infl ammatory cells or macrophages surrounded by lymphocytes [5]. It is also known that salivary glands NK cells, although related to conventional NK cells, are a distinct subset of innate lymphoid cells that deviates from the conventional developmental pathway, perhaps under the infl uence of tissue-specific factors [4]. Improved the lymphocytes population infl uence on a formation of different organs and systems. Immature lymphocytes migrated from thymus to the peripheral organs of immune system [9]. One of the determinatives that result in violation of major salivary glands morphogenesis and as a result the development of its pathology is the condition of pregnant women health, more than half of them have chronic diseases and system functional disorders which are accompanied by the immune pathological condition, namely by antigen infl uence on a fetus [3,9]. In the literature there are no systematic data concerning the intrauterine antigen infl uence on morphogenesis of major UDC: 611.316.018.53:616-097.1]-053.31-092.9

PNA- and SBA-positive lymphocytes content in the major salivary glands’ structures during early postnatal period after intrauterine antigenic action