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We have used an insertional mutagenesis approach to generate new  C .  reinhardtii  motility mutants. Of 56 mutants isolated, one is a new allele at the  ODA3  locus, called  oda3-6 . Similar to the previously characterized  oda3  alleles,  oda3-6  has a slow-jerky swimming phenotype and reduced swimming speed. The  oda3-6  mutant fails to assemble the outer dynein arm motor and outer dynein arm—docking complex (ODA-DC) in the ciliary axoneme due to an insertion in the 5’ end of the  DCC1  gene, which encodes the DC1 subunit of the ODA-DC. Transformation of  oda3-6  with the wild-type  DCC1  gene rescues the mutant swimming phenotype and restores assembly of the ODA-DC and the outer dynein arm in the cilium. This is the first  oda3  mutant to be characterized at the molecular level and is likely to be very useful for further analysis of DC1 function.

Characterization of a new oda3 allele, oda3-6, defective in assembly of the outer dynein arm-docking complex in Chlamydomonas reinhardtii