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Resistance to biotrophic pathogens is largely dependent on the hormone salicylic acid (SA) while jasmonic acid (JA) regulates resistance against necrotrophs. JA negatively regulates SA and is, in itself, negatively regulated by SA. A key component of the JA signal transduction pathway is its receptor, the  COI1  gene. Mutations in this gene can affect all the JA phenotypes, whereas mutations in other genes, either in JA signal transduction or in JA biosynthesis, lack this general effect. To identify components of the part of the resistance against biotrophs independent of SA, a mutagenised population of  NahG  plants (severely depleted of SA) was screened for suppression of susceptibility. The screen resulted in the identification of intragenic and extragenic suppressors, and the results presented here correspond to the characterization of one extragenic suppressor,  coi1 -40.  coi1 -40 is quite different from previously described  coi1  alleles, and it represents a strategy for enhancing resistance to biotrophs with low levels of SA, likely suppressing  NahG  by increasing the perception to the remaining SA. The phenotypes of  coi1 -40 lead us to speculate about a modular function for COI1, since we have recovered a mutation in  COI1  which has a number of JA-related phenotypes reduced while others are equal to or above wild type levels.

An Allele of Arabidopsis COI1 with Hypo- and Hypermorphic Phenotypes in Plant Growth, Defence and Fertility