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NAC ( N AM,  A TAF1-2, and  C UC2) proteins constitute one of the largest families of plant-specific transcription factors and have been shown to be involved in diverse plant processes including plant growth, development, and stress-tolerance. In this study, a stress-responsive  NAC  gene,  EcNAC1 , was isolated from the subtracted stress cDNA library generated from a drought adapted crop, finger millet, and characterized for its role in stress-tolerance. The expression analysis showed that  EcNAC1  was highly induced during water-deficit and salt stress.  EcNAC1  shares high amino acid similarity with rice genes that have been phylogenetically classified into stress-related  NAC  genes. Our results demonstrated that tobacco transgenic plants expressing  EcNAC1  exhibit tolerance to various abiotic stresses like simulated osmotic stress, by polyethylene glycol (PEG) and mannitol, and salinity stress. The transgenic plants also showed enhanced tolerance to methyl-viologen (MV) induced oxidative stress. Reduced levels of reactive oxygen species (ROS) and ROS-induced damage were noticed in pot grown transgenic lines under water-deficit and natural high light conditions. Root growth under stress and recovery growth after stress alleviation was more in transgenic plants. Many stress-responsive genes were found to be up-regulated in transgenic lines expressing  EcNAC1.  Our results suggest that  EcNAC1  overexpression confers tolerance against abiotic stress in susceptible species, tobacco.

Expression of a Finger Millet Transcription Factor, EcNAC1, in Tobacco Confers Abiotic Stress-Tolerance