English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

The trauma-hemorrhage- induced immunosuppression is also associated with an increased susceptibility to subsequent sepsis, organ failure and mortality. Previous studies demonstrated that trauma-hemorrhage induces immunosuppression in both innate immune systems and adaptive immune systems. The in vitro capacity of peritoneal, alveolar and splenic macrophages to release interleukin (IL)-1, IL-6 and tumour necrosis factor (TNF)-α in response to lipopolysaccharide (LPS) stimulation is diminished after traumahemorrhage. In contrast, Kupffer cells have been shown to have an enhanced capacity to produce pro-inflammatory

oa anaesthetics

Trauma-hemorrhage and dendritic cell functions: A critical review of splenic dendritic cell dysfunction following trauma-hemorrhage and therapeutic approach