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Pseudohypoaldosteronism (PHA) comprises of a heterogeneous group of disorders of electrolyte metabolism characterized by an apparent state of renal tubular unresponsiveness or resistance to the action of aldosterone. It is manifested by presence of salt wasting, hyperkalemia, and metabolic acidosis [1]. Signs and symptoms of PHA can be rather unspecific and include poor feeding, poor weight gain or failure to thrive, vomiting, diarrhea, polyuria, and dehydration [2]. PHA is classified as primary and secondary type. Secondary transient PHA has been reported in infants with urinary tract infection (UTI) and urinary tract malformation. The acute biochemical presentation of PHA is identical to adrenal crisis due to congenital adrenal hyperplasia (CAH) and at initial stages the condition is suspected as CAH. Here, we report an infant who presented with failure to thrive and finally diagnosed with transient PHA due to UTI with vesicoureteral reflux.

Transient Pseudohypoaldosteronism in a 5-Month-old Infant Manifested as a Failure to Thrive