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There are several reports that cadmium added to the diet of experimental animals is concentrated in the liver and kidneys1-3). Once cadmium enters the body, its excretion is extremely slow, and the biological half times of cadmium in the kidney and liver are longer than in other organs4). Other reports indicate that the accumulation rate of orally administered cadmium becomes lower in proportion to the period of ingestion. Tsuchiya and Seki5) (1971) reported that the cadmium accumulated in mouse kidney and liver only increased slightly from 2 to 3 month intake. Therefore, the gastrointestinal tract may play an important role in protecting the body against the noxious effects of dietary cadmium. Numerous factors influence cadmium absorption from the intestine6-8). When cadmium is orally to experimental animals with other metals, they influence the absorption of cadmium. For example, it has been reported that Hg reduces cadmium absorption and that Zn increases it9). Valberg et al.10) (1976) found that mice raised on an iron deficient diet showed an augmented absorption of cadmium. The present study investigated the absorption rate of 109Cd administered to mice previous treated with an oral dose of cadmium for set periods.

The metabolism of 109Cd administered to mice previously given an oral dose of cadmium.