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procedures.1 Both cardiac troponins and creatine kinase MB are sensitive and specific biomarkers for quantitative diagnosis of irreversible myocardial injury, and the release of these biomarkers is associated with increased risk of death and myocardial infarction (MI).1,2 A previous report showed that a 5-fold post-procedural elevation of cardiac troponin T above normal levels is an independent predictor of a composite of death, MI, and revascularization at 1 year (hazard ratio, 2.39; 95% confidence interval, 1.09–5.26).3 Importantly, because PMI also occurs in a considerable P ercutaneous coronary intervention (PCI) is established as one of the most common procedures for the treatment of coronary artery disease (CAD), based on considerable evidence. The advances in procedural techniques, stent materials and aggressive antiplatelet therapy have decreased the incidence of major periprocedural complications of PCI. However, periprocedural myocardial injury (PMI) is still one of the inevitable complications of PCI, resulting from distal embolization, side-branch occlusion, coronary dissection and disruption of collateral flow. A previous study has reported several risk factors for PMI, including more extensive disease, multivessel disease, complex lesion morphology, coronary thrombus and calcification, left ventricular systolic dysfunction and urgent Article p ????

Prevention of Periprocedural Myocardial Injury During Percutaneous Coronary Intervention in Patients With Stable Coronary Artery Disease