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Atherosclerosis is characterized as a chronic inflammatory disease. The complex network of both innate and adaptive immunity plays a significant role in the development and progression of atherosclerotic plaques. Rupture or erosion of these uring the past decades several attempts have been made to reduce cardiovascular morbidity and mortality. Unfortunately, a high disease burden still covers the developed world and is rapidly emerging in the developing countries, with all the characteristics of an epidemic. Despite intensive work in trying to prevent post-event pathological remodeling and many efforts made to improve patients’ outcomes, the data are sobering.1,2 Therefore, prevention of major D

Treating the Unstable Atherosclerotic Plaque by Targeting Activated Factor X – Anticoagulation and Beyond –