English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Left (L) renal perfusion with an aminonucleoside of puromycin (PA), was used to produce unilateral proteinuria in 15 rats to examine the mechanisms responsible for renal salt retention in the nephrotic syndrome. Thirteen control rats underwent L renal perfusion with isotonic saline. Animals were studied 8 (group I) or 13 (group II) days after perfusion. Renal perfusion with saline per se did not change the glomerular filtration rate, renal plasma flow, or absolute and fractional excretion of sodium (Na) from the perfused kidney. PA animals showed a significant decrease in glomerular filtration rate from the perfused kidney and a proportional decrease in the absolute excretion of Na from the PA perfused kidney as compared to the right kidney. The fractional excretion of Na was equivalent in the L and R kidneys of the PA animals. The mean absolute Na excretion from the nonproteinuric R kidney of PA rats was almost twice that of the R kidney of the controls. The increased Na excretion by the nonproteinuric kidney of the PA animals compensated for the sodium retention by the proteinuric kidney.

Renal Function in Rats with Unilateral Proteinuria Produced by Renal Perfusion with Aminonucleoside