English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Objectives  Earlier work in our lab identified a spontaneous mutant (  l   ike   s   unn   s   upernodulator - ls s) in  Medicago truncatula,  resulting in increased nodulation. Molecular genetic evidence indicated the phenotype was due to an unknown lesion resulting in  cis -silencing of the  SUNN  gene. Altered methylation of the promoter was suspected, but analysis of the  SUNN  promoter by bisulfite sequencing at the time of publication revealed no significant methylation differences between the  SUNN  promoter in wild type and  lss  plants. Using advances in methylome generation we compared the methylome of wild type and the  lss  mutant in the larger 810 kB area of the genome where  lss  maps.    Data description  The data show the distribution of types of methylation across the entire genome between A17 wild type and  lss  mutants, the number of differentially methylated cytosines between genotypes, and the overall pattern of gene methylation between genotypes. We expect the wild type data will be especially useful as a reference for other investigations of methylation using  M. truncatula .

Whole genome bisulfite sequencing of Medicago truncatula A17 wild type and lss mutants