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Background  Moose ( Alces alces ) are a culturally and economically valued species in Minnesota. However, the moose population has experienced a sudden, marked decline in their range, including extirpation in the northwest and a 66% decline in the last decade in the northeast portions of the state. Although the exact cause of this decline is unclear, parasitic metastrongylid and filarioid nematode infections are known causes of morbidity and mortality in moose across North America.    Methods  To determine if these parasitic nematodes could be contributing to the Minnesota moose population decline, we molecularly examined banked tissues obtained from moose that died of known and unknown causes for the presence of nematode DNA. Extracted brain DNA of 34 individual moose was amplified utilizing primers targeting the  18S  rRNA gene and internal transcribed spacer regions of nematodes.    Results  DNA sequencing revealed that PCR products obtained from 15 (44.1%) of the moose were 99% identical to  Parelaphostrongylus tenuis , a metastrongylid known to cause neurological disease and death. Additionally, brain tissue from 20 (58.8%) individuals yielded sequences that most closely aligned with  Elaeophora schneideri , a parasite associated with neurological impairment but previously unreported in Minnesota.  Setaria yehi , a common filarioid parasite of deer, was also detected in the brain tissue of 5 (14.7%) moose. Molecular screening of 618 captured tabanid flies from four trapping sites revealed  E. schneideri  was present (6%) in the Minnesota environment and transmission could occur locally. Prevalence rates among the flies ranged between 0–100% per trapping site, with  Chrysops  spp. and  Hybomitra  spp. implicated as the vectors .Conclusions  Ultimately, these data confirm that  P. tenuis  is widespread in the Minnesota moose population and raises the question of the significance of  E. schneideri  as a contributing factor to morbidity and mortality in moose.    Electronic supplementary material  The online version of this article (10.1186/s13071-018-3077-0) contains supplementary material, which is available to authorized users.

Emergence of the arterial worm Elaeophora schneideri in moose (Alces alces) and tabanid fly vectors in northeastern Minnesota, USA