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Sphingosine-1-phosphate (S1P) regulates distinct biological functions by binding to S1P receptors S1PR1-5 and is generated by phosphorylation of sphingosine via sphingosine kinases (SPHK) 1 and 2. The S1P mimetic FTY720 acts in its phosphorylated isoform as an unselective agonist on S1PR1 and S1PR3-5 and a selective functional antagonist on S1PR1 ([1]). The role of the S1P-S1PR signalling axis in the pathophysiology of septic cardiomyopathy is not known.

Enhanced sphingosine-1-phosphate levels ameliorate murine septic cardiomyopathy