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Ventilator-induced lung injury (VILI) has been related not only to pulmonary injury but also to systemic damage. We performed a bioassay using ex vivo models of VILI and of vascular function in order to determine the role of pulmonary-derived factors in ventilator-induced endothelial dysfunction. The involvement of nitro-oxidative stress was also examined [1].

intensive care medicine experimental

0984. Perfusate from lungs ventilated ex-vivo with high tidal volumen induce in vitro endotelial dysfunction reversed by superoxide dismutase and tempol