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Receptor for advanced glycation endproducts (RAGE) is a transmembrane pattern-recognition receptor of the immunoglobulin superfamily that is abundantly expressed in the lung and primarily located on the basal surface of alveolar type I cells. RAGE is implicated in ARDS as an important pathway to alveolar inflammation and, when its soluble form sRAGE is assayed in plasma or pulmonary edema fluid, as a marker of AT I cell injury [1]. Functional activity of AT 1 cells can be assessed by the measurement of alveolar fluid clearance (AFC) rate [2], but the relationship between sRAGE plasma levels of sRAGE and AFC rates has never been investigated.

intensive care medicine experimental

0853. Elevated levels of soluble rage predict impaired alveolar fluid clearance in a translational mouse model of acute respiratory distress syndrome (ARDS)