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Adipose tissue plays an intriguing role in the endocrine system by producing adipokines [1]. In patients with obesity, metabolic syndrome, and type 2 diabetes (DM2), insulin resistance is influenced by adipokines [1].  From a metabolic point of view, the stress response caused by surgical injury imitates the diabetic state. Stereotypical endocrine-metabolic and inflammatory reactions result in catabolism and pronounced insulin resistance, which is related to outcome [2].

0714. Interaction between adipokines and the metabolic stress response: angpt l2 cxcl5 and visfatin in patients undergoing cardiac surgery