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Aims Hypoxic-ischemic injury (HI) to the developing brain remains a major cause of morbidity. Hypothermia is currently the only established neuroprotective treatment available for term borns with hypoxic-ischemic encephalopathy, saving one in eight infants from developing severe neurological deficits. Therefore, additional treatments with clinically applicable drugs are indispensable. Furthermore, the pathophysiological mechanisms of hypothermia-induced recovery are not clearly understood. This study examines a potential additive neuroprotective effect of hypothermia combined with levetiracetam in neonatal mouse HI.

Neuroprotective effects of hypothermia and levetiracetam after hypoxia-ischemia in the neonatal mouse brain