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Background There are nitric oxide (NO)-sensitive and -insensitive forms of soluble guanylyl cyclase (sGC). This balance is shifted to the latter under stress conditions associated with increased production of reactive oxygen species (ROS) [1,2]. The present study investigated whether smoking, a well-documented source of ROS, affects NO-sensitive and -insensitive sGC-mediated effects in human arteries.

Influence of smoking on vascular reactivity to cGMP generators in human internal thoracic arteries