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Background The endothelium plays a fundamental role in maintaining vascular homeostasis by releasing factors that regulate local blood flow, systemic blood pressure, and the reactivity of leukocytes and platelets. Accordingly, endothelial dysfunction underpins many cardiovascular diseases, including hypertension, myocardial infarction and stroke. C-type natriuretic peptide (CNP) is a paracrine mediator that possesses a unique vasoand cardioprotective pharmacodynamic profile; however, there is a paucity of information regarding a physiological role for CNP within the cardiovascular system.

Pivotal vascular homeostatic role for endothelium-derived C-type natriuretic peptide (CNP)