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Methods The following variables were collected from 59 HIV+ subjects attending our outpatient department between Jan. 2007 and June 2008: traditional CV risk factors; time and type of exposure to cART; genetic polymorphisms associated with inflammation and lipodystrophy (ApoE, LPL, MDR1 3435, TNF238 and 308, adiponectin 45 and 276); endothelial adhesion molecules and platelet activation markers (ICAM-1, t-PA, PAI-1, P-selectin); M-mode carotid duplex ultrasound to measure intima-media thickness (IMT, normal value <0.6 mm), distensibility index (DI, normal value > 0.41 mm), presence of plaques (IMT > 1 mm).

Do genetic polymorphisms associated with inflammation/lipodystrophy or endothelial damage predict carotid alterations in HIV+ subjects under cART?