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Ying Yang,1,5 Xiaogang Shu,2,3,5 Dan Liu,1,5 You Shang,2,3 YanWu,2,3 Lei Pei,1 Xin Xu,3 Qing Tian,1 Jian Zhang,3 KunQian,3 Ya-Xian Wang,4 Ronald S. Petralia,4 Weihong Tu,3 Ling-Qiang Zhu,1,* Jian-Zhi Wang,1,* and Youming Lu1,3,* 1Key Laboratory of Neurological Disease, Ministry of Education, China, and Department of Pathophysiology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, China 2Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, China 3Neuroscience Center of Excellence and Department of Neurology, Louisiana State University School of Medicine, New Orleans, LA 70112, USA 4Laboratory of Neurochemistry, National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, MD 20892, USA 5These authors contributed equally to this work *Correspondence: zhulq@mail.hust.edu.cn (L.-Q.Z.), wangjz@mail.hust.edu.cn (J.-Z.W.), lym@mail.hust.edu.cn (Y.L.) DOI 10.1016/j.neuron.2012.02.003

EPAC null mutation impairs learning and social interactions via aberrant regulation of miR-124 and Zif268 translation