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Parkinson's disease (PD) comprises a range of motor andcognitive disturbances, a portion of which respond little,if at all, to levodopa treatment and are therefore consid-ered to be due to nondopaminergic lesions [1,2]. Moresevere cognitive impairments and higher risk for develop-ing dementia have been associated with the clinical man-ifestations of postural instability and gait disorder whichare characterized by refractoriness to levodopa [2,3].

Clinical correlates of mental dysfunction in Parkinson's disease without dementia