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Background It is well established that cytokines signal the brain and serve as mediators between immune and nerve cells. Recent research findings have led to speculation that the neuroimmunologic consequences of cytokine levels might be involved in the pathogenesis of depression. Although TNF-a is required for normal immune responses, its overexpression has severe pathological consequences. The present study was undertaken to determine whether TNF-a secretion levels before and after mitogenic stimulation differ between depressed patients and matched healthy controls during clinical improvement.

Association between TNF-a secretion levels in major depressive disorder and symptoms' severity, before and after mitogen stimulation