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Background Patients with mild cognitive impairment (MCI) is reported to develop Alzheimer's disease (AD) at the rate of 12% per year, greatly exceeding the 1% to 2% incidence of normal controls. Several studies have shown an increase in plasma Aβ42 in MCI compared to normal and AD patients. The efficiency of Aβ peptides elimination in earlier stages of AD has proven in animal models. We found no study measuring phospho-tau (p-tau) level in plasma.

High plasma Amyloid β42 and P-tau in mild cognitive impairment as risk factors of the disease