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Background During simian immunodeficiency virus (SIV) infection changes occur in the gd TCR T cell population. The major subset in mucosal tissue, Vd1, becomes prevalent in peripheral blood relative to the Vd2 subset. gd T cells have the ability to expand in vitro and in vivo making them attractive as cytotoxic effectors against infections like SIV and HIV.

Changes in gd T cell function and gut homing receptors following SIV infection of rhesus macaques