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Background African green monkeys (AGMs), natural SIV hosts, sustain nonpathogenic infections and rarely transmit the virus to their suckling infants, despite exposure to high milk virus RNA loads. Furthermore, we previously reported strong autologous neutralization responses in milk of SIV-infected AGMs which could contribute to impediment of infant virus acquisition. Comparing mucosal B cell populations and responses in milk of AGMs to that of rhesus monkeys (RMs), symptomatic SIV hosts with high rates of postnatal transmission, could elucidate the protection against postnatal virus transmission in natural SIV hosts. Methods

Strong SIV gp120-specific IgG/IgA responses in milk of African green monkeys may contribute to the rarity of postnatal transmission in this species