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Background Multiple sclerosis (MS) is considered a multifactorial disease with both genetic and environmental components in the etiology. Certain demyelinating diseases in other species have been shown to be caused by retroviruses, and the possibility remains that also MS has a viral component. As there is little evidence that MS is contagious, we have focused our attention on the human endogenous retroviruses, which are transmitted vertically from parent to offspring as Mendelian loci.

Involvement of the endogenous retroviral locus HERV-Fc1 on the human X-chromosome in multiple sclerosis