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Background of Study HIV infection accelerates the rate of HCV progression to fibrosis which represents the main factor affecting the prognosis of hepatitis C as well the best indicator of disease status. There is increasing evidence that liver fibrosis is a dynamic pathology c process in which the altered balance between matrix metalloproteiases (MMPs) and their specific inhibitors (TIMPs) may play a major role.

Liver disease progression in HIV/HCV co-infected patients: a role for metalloproteinases and their specific inhibitors